Anavar & Test
Started By konan, Aug 28 2011 06:53 PM
11 replies to this topic
#1
Posted 28 August 2011 - 06:53 PM
I am thinking running a Anavar & Test E cycle. I am looking to add some mass and still have a chiseled mid section. I am not looking to blow up over night as a do when i run Test E with Dbol(water retention). I have read the Anavar 101 post. A source out there carries GP oxan(anavar) . In their description they state that "VAR is a mild steriod" I am chosing Test E over Test P so i dont have to stick myself as much. What are your thoughts on my choice
#2
Posted 28 August 2011 - 09:39 PM
I ran Anavar for 8 weeks 50mg a day and was not impressed at all. I hope you get a good batch. I won't ever run it again. just to expensive. I like GH better for that kind of expense. Good luck.
#3
Posted 29 August 2011 - 03:11 AM
I am thinking running a Anavar & Test E cycle. I am looking to add some mass and still have a chiseled mid section. I am not looking to blow up over night as a do when i run Test E with Dbol(water retention). I have read the Anavar 101 post. A source out there carries GP oxan(anavar) . In their description they state that "VAR is a mild steriod" I am chosing Test E over Test P so i dont have to stick myself as much. What are your thoughts on my choice
If you can give us more info it would be more helpful. What were your previous cycles, what are current stats?
TBH why would you run such a mild steroid like Var alongside test?
If you don't want bloat why not take tren in with your cycle, I'm a huge, huge fan of test & tren cycles.
#4
Posted 29 August 2011 - 05:11 AM
I am thinking running a Anavar & Test E cycle. I am looking to add some mass and still have a chiseled mid section. I am not looking to blow up over night as a do when i run Test E with Dbol(water retention). I have read the Anavar 101 post. A source out there carries GP oxan(anavar) . In their description they state that "VAR is a mild steriod" I am chosing Test E over Test P so i dont have to stick myself as much. What are your thoughts on my choice
If you can give us more info it would be more helpful. What were your previous cycles, what are current stats?
TBH why would you run such a mild steroid like Var alongside test?
If you don't want bloat why not take tren in with your cycle, I'm a huge, huge fan of test & tren cycles.
Thanx Cowboy.
I have had great success in the past on a Test E,Dbol 10mg and Tren @ 15wks. I am currently 190lbs BF approx 10% 5ft 9. I took a break from gym early spring til now. In that time frame i lost a total of 35lbs(muscle mass included).Even though i was out of the gym i was still involved in some very vigorous outdoor activities. I am ready to get back but i do not want to blow up overnight and have have people raising their eyebrows. That is why i was choosing Anavar over dbol. in that sense that i still want some gains but 2 the very same time not be the talk of the town.
#5
Posted 29 August 2011 - 05:23 AM
What about just doing a 6 week Tbol cycle at 60mg? This way you're gonna keep BF down as well as put on some good size without it being too obvious.
#6
Posted 29 August 2011 - 09:07 PM
What about just doing a 6 week Tbol cycle at 60mg? This way you're gonna keep BF down as well as put on some good size without it being too obvious.
I haven't Ben around the board for a while. Was that supposed to be dbo 6wk cycle @ 60mg/day. How can I incorporate trek into the mix /
#7
Posted 30 August 2011 - 03:47 AM
Turinabol/Tbol:
You'll get a good run outta this at 60mg ED, I also throw in Proviron at 50mg a day as well.
QUOTE
Oral Turinabol (4-chlorodehydromethyltestosterone)
Androgenic no data available
Anabolic >100
Standard Methyltestosterone (oral)
Chemical Names 4-chloro-17a-methyl-17b-hydroxyandrosta-1,4-dien-3-one
Estrogenic Activity none
Progestational Activity no data available (low)
Description:
Chlorodehydromethyltestosterone is a potent derivative of Dianabol. This oral steroid is structurally a cross between methandrostenolone and clostebol (4-chlorotestosterone), having the same base structure as Dianabol with the added 4-chloro alteration of clostebol. This alteration makes chlorodehydromethyltestosterone a milder cousin of Dianabol, the new steroid displaying no estrogenic and much less androgenic activity in comparison to its more famous counterpart. The anabolic activity chlorodehydromethyltestosterone is somewhat lower than that of Dianabol as well, but it does maintain a much more favorable balance of anabolic to androgenic effect. This means that at any given level of muscle-building activity, chlorodehydromethyltestosterone will be less likely to produce androgenic side effects.
Structural Characteristics:
Chlorodehydromethyltestosterone is a modified form of testosterone. It differs by: 1) the addition of a methyl group at carbon 17-alpha to protect the hormone during oral administration 2) the introduction of a double bond between carbons 1 and 2 (1-ene), which shifts the anabolic to androgenic ratio in favor of the former, and 3) the attachment of a chloro group at carbon 4, which inhibits steroid aromatization and reduces relative androgenicity.
Side Effects (Estrogenic):
Chlorodehydromethyltestosterone is not aromatized by the body, and is not measurably estrogenic. An anti-estrogen is not necessary when using this steroid, as gynecomastia should not be a concern even among sensitive individuals. Since estrogen is the usual culprit with water retention, this steroid instead produces a lean, quality look to the physique with no fear or excess subcutaneous fluid retention. This makes it a favorable steroid to use during cutting cycles, when water and fat retention are major concerns.
Side Effects (Androgenic):
Although chlorodehydromethyltestosterone is classified as an anabolic steroid, androgenic side effects are still possible with this substance. These may include bouts of oily skin, acne, and body/facial hair growth. Doses higher than normally prescribed are more likely to cause such side effects. Anabolic/androgenic steroids may also aggravate male pattern hair loss. Women are also warned of the potential virilizing effects of anabolic/androgenic steroids. These may include a deepening of the voice, menstrual irregularities, changes in skin texture, facial hair growth, and clitoral enlargement. Chlorodehydromethyltestosterone is not extensively metabolized by the 5-alpha reductase enzyme, so its relative androgenicity is not greatly altered by the concurrent use of finasteride.
Side Effects (Hepatotoxicity):
Chlorodehydromethyltestosterone is a c17-alpha alkylated compound. This alteration protects the drug from deactivation by the liver, allowing a very high percentage of the drug entry into the bloodstream following oral adminstration. C17-alpha alkylated anabolic/androgenic steroids can be hepatotoxic. Prolonged or high exposure may result in liver damage. In rare instances life threatening dysfunction may develop. It is advisable to visit a physician periodically during each cycle to monitor liver function and overall health. Intake of c17-alpha alkylated steroids is commonly limited to 6 - 8 weeks, in an effort to avoid escalating liver strain.
Side Effects (Cardiovascular):
Anabolic/androgenic steroids can have deleterious effects on serum cholesterol. This includes a tendancy to reduce HDL (good) cholesterol values and increase LDL (bad) cholesterol values, which may shift the HDL to LDL balance in a direction that favors greater risk of arteriosclerosis. The relative impact of an anabolic/androgenic steroid on serum lipids is dependent on the dose, route of administration (oral vs. injectable) type of steroid (aromatizable or non-aromatizable), and level of resistance to hepatic metabolism. Chlorodehydromethyltestosterone has a strong effect on the hepatic management of cholesterol due to its structural resistance to liver breakdown, non-aromatizable nature, and route of administration. Anabolic/androgenic steroids may also adversely affect blood pressure and triglycerides, reduce endothelial relaxation, and support left ventricular hypertrophy, all potentially increasing the risk of cardiovascular disease and myocardial infarction.
Side Effects (Testosterone Suppression):
All anabolic/androgenic steroids when taken in doses sufficient to promote muscle gain are expected to suppress endogenous testosterone production. Without the intervention of testosterone-stimulating substancestestosterone levels should return to normal within 1 - 4 months of drug secession. Note that prolonged hypogonadotrophic hypogonadism can develop secondary to steroid abuse, necessitating medical intervention.
Administration (Men):
A common clinical dose of chlorodehydromethyltestosterone is estimated to be 5mg per day; actual prescribing guidelines are unavailable. In the athletic arena, an effective oral daily dosage falls in the range of 15 - 40mg, taken in cycles lasting no more than 6 - 8 weeks to minimize hepatotoxicity. This level is sufficient for measurable increases in lean muscle mass and strength. This agent is most often applied as a pre-contest or cutting steroid for bodybuilding purposes, and is not viewed as an ideal bulking agent due to lack of estrogenicity. Athletes in sports where speed tends to be a primary focus also fine strong favor in chlorodehydromethyltestosterone, obtaining a strong anabolic benefit without having to carry around any extra water or fat weight.
Administration (Woman):
A common clinical dose of chlorodehydromethyltestosterone is estimated to be 1 - 2.5mg per day; actual prescribing guidelines are unavailable. In the athletic arena, women would commonly take a single 5mg tablet per day, taken in cycles lasting no more than 4 - 6 weeks to minimize hepatotoxicity. Virilizing effects are unlikely at this level of use. Much higher doses often used with female athletes in the former GDR doping program, but often to detriment of strong virilizing side effects.
Androgenic no data available
Anabolic >100
Standard Methyltestosterone (oral)
Chemical Names 4-chloro-17a-methyl-17b-hydroxyandrosta-1,4-dien-3-one
Estrogenic Activity none
Progestational Activity no data available (low)
Description:
Chlorodehydromethyltestosterone is a potent derivative of Dianabol. This oral steroid is structurally a cross between methandrostenolone and clostebol (4-chlorotestosterone), having the same base structure as Dianabol with the added 4-chloro alteration of clostebol. This alteration makes chlorodehydromethyltestosterone a milder cousin of Dianabol, the new steroid displaying no estrogenic and much less androgenic activity in comparison to its more famous counterpart. The anabolic activity chlorodehydromethyltestosterone is somewhat lower than that of Dianabol as well, but it does maintain a much more favorable balance of anabolic to androgenic effect. This means that at any given level of muscle-building activity, chlorodehydromethyltestosterone will be less likely to produce androgenic side effects.
Structural Characteristics:
Chlorodehydromethyltestosterone is a modified form of testosterone. It differs by: 1) the addition of a methyl group at carbon 17-alpha to protect the hormone during oral administration 2) the introduction of a double bond between carbons 1 and 2 (1-ene), which shifts the anabolic to androgenic ratio in favor of the former, and 3) the attachment of a chloro group at carbon 4, which inhibits steroid aromatization and reduces relative androgenicity.
Side Effects (Estrogenic):
Chlorodehydromethyltestosterone is not aromatized by the body, and is not measurably estrogenic. An anti-estrogen is not necessary when using this steroid, as gynecomastia should not be a concern even among sensitive individuals. Since estrogen is the usual culprit with water retention, this steroid instead produces a lean, quality look to the physique with no fear or excess subcutaneous fluid retention. This makes it a favorable steroid to use during cutting cycles, when water and fat retention are major concerns.
Side Effects (Androgenic):
Although chlorodehydromethyltestosterone is classified as an anabolic steroid, androgenic side effects are still possible with this substance. These may include bouts of oily skin, acne, and body/facial hair growth. Doses higher than normally prescribed are more likely to cause such side effects. Anabolic/androgenic steroids may also aggravate male pattern hair loss. Women are also warned of the potential virilizing effects of anabolic/androgenic steroids. These may include a deepening of the voice, menstrual irregularities, changes in skin texture, facial hair growth, and clitoral enlargement. Chlorodehydromethyltestosterone is not extensively metabolized by the 5-alpha reductase enzyme, so its relative androgenicity is not greatly altered by the concurrent use of finasteride.
Side Effects (Hepatotoxicity):
Chlorodehydromethyltestosterone is a c17-alpha alkylated compound. This alteration protects the drug from deactivation by the liver, allowing a very high percentage of the drug entry into the bloodstream following oral adminstration. C17-alpha alkylated anabolic/androgenic steroids can be hepatotoxic. Prolonged or high exposure may result in liver damage. In rare instances life threatening dysfunction may develop. It is advisable to visit a physician periodically during each cycle to monitor liver function and overall health. Intake of c17-alpha alkylated steroids is commonly limited to 6 - 8 weeks, in an effort to avoid escalating liver strain.
Side Effects (Cardiovascular):
Anabolic/androgenic steroids can have deleterious effects on serum cholesterol. This includes a tendancy to reduce HDL (good) cholesterol values and increase LDL (bad) cholesterol values, which may shift the HDL to LDL balance in a direction that favors greater risk of arteriosclerosis. The relative impact of an anabolic/androgenic steroid on serum lipids is dependent on the dose, route of administration (oral vs. injectable) type of steroid (aromatizable or non-aromatizable), and level of resistance to hepatic metabolism. Chlorodehydromethyltestosterone has a strong effect on the hepatic management of cholesterol due to its structural resistance to liver breakdown, non-aromatizable nature, and route of administration. Anabolic/androgenic steroids may also adversely affect blood pressure and triglycerides, reduce endothelial relaxation, and support left ventricular hypertrophy, all potentially increasing the risk of cardiovascular disease and myocardial infarction.
Side Effects (Testosterone Suppression):
All anabolic/androgenic steroids when taken in doses sufficient to promote muscle gain are expected to suppress endogenous testosterone production. Without the intervention of testosterone-stimulating substancestestosterone levels should return to normal within 1 - 4 months of drug secession. Note that prolonged hypogonadotrophic hypogonadism can develop secondary to steroid abuse, necessitating medical intervention.
Administration (Men):
A common clinical dose of chlorodehydromethyltestosterone is estimated to be 5mg per day; actual prescribing guidelines are unavailable. In the athletic arena, an effective oral daily dosage falls in the range of 15 - 40mg, taken in cycles lasting no more than 6 - 8 weeks to minimize hepatotoxicity. This level is sufficient for measurable increases in lean muscle mass and strength. This agent is most often applied as a pre-contest or cutting steroid for bodybuilding purposes, and is not viewed as an ideal bulking agent due to lack of estrogenicity. Athletes in sports where speed tends to be a primary focus also fine strong favor in chlorodehydromethyltestosterone, obtaining a strong anabolic benefit without having to carry around any extra water or fat weight.
Administration (Woman):
A common clinical dose of chlorodehydromethyltestosterone is estimated to be 1 - 2.5mg per day; actual prescribing guidelines are unavailable. In the athletic arena, women would commonly take a single 5mg tablet per day, taken in cycles lasting no more than 4 - 6 weeks to minimize hepatotoxicity. Virilizing effects are unlikely at this level of use. Much higher doses often used with female athletes in the former GDR doping program, but often to detriment of strong virilizing side effects.
You'll get a good run outta this at 60mg ED, I also throw in Proviron at 50mg a day as well.
#8
Posted 30 August 2011 - 07:56 AM
Turinabol/Tbol:
QUOTE
Oral Turinabol (4-chlorodehydromethyltestosterone)
Androgenic no data available
Anabolic >100
Standard Methyltestosterone (oral)
Chemical Names 4-chloro-17a-methyl-17b-hydroxyandrosta-1,4-dien-3-one
Estrogenic Activity none
Progestational Activity no data available (low)
Description:
Chlorodehydromethyltestosterone is a potent derivative of Dianabol. This oral steroid is structurally a cross between methandrostenolone and clostebol (4-chlorotestosterone), having the same base structure as Dianabol with the added 4-chloro alteration of clostebol. This alteration makes chlorodehydromethyltestosterone a milder cousin of Dianabol, the new steroid displaying no estrogenic and much less androgenic activity in comparison to its more famous counterpart. The anabolic activity chlorodehydromethyltestosterone is somewhat lower than that of Dianabol as well, but it does maintain a much more favorable balance of anabolic to androgenic effect. This means that at any given level of muscle-building activity, chlorodehydromethyltestosterone will be less likely to produce androgenic side effects.
Structural Characteristics:
Chlorodehydromethyltestosterone is a modified form of testosterone. It differs by: 1) the addition of a methyl group at carbon 17-alpha to protect the hormone during oral administration 2) the introduction of a double bond between carbons 1 and 2 (1-ene), which shifts the anabolic to androgenic ratio in favor of the former, and 3) the attachment of a chloro group at carbon 4, which inhibits steroid aromatization and reduces relative androgenicity.
Side Effects (Estrogenic):
Chlorodehydromethyltestosterone is not aromatized by the body, and is not measurably estrogenic. An anti-estrogen is not necessary when using this steroid, as gynecomastia should not be a concern even among sensitive individuals. Since estrogen is the usual culprit with water retention, this steroid instead produces a lean, quality look to the physique with no fear or excess subcutaneous fluid retention. This makes it a favorable steroid to use during cutting cycles, when water and fat retention are major concerns.
Side Effects (Androgenic):
Although chlorodehydromethyltestosterone is classified as an anabolic steroid, androgenic side effects are still possible with this substance. These may include bouts of oily skin, acne, and body/facial hair growth. Doses higher than normally prescribed are more likely to cause such side effects. Anabolic/androgenic steroids may also aggravate male pattern hair loss. Women are also warned of the potential virilizing effects of anabolic/androgenic steroids. These may include a deepening of the voice, menstrual irregularities, changes in skin texture, facial hair growth, and clitoral enlargement. Chlorodehydromethyltestosterone is not extensively metabolized by the 5-alpha reductase enzyme, so its relative androgenicity is not greatly altered by the concurrent use of finasteride.
Side Effects (Hepatotoxicity):
Chlorodehydromethyltestosterone is a c17-alpha alkylated compound. This alteration protects the drug from deactivation by the liver, allowing a very high percentage of the drug entry into the bloodstream following oral adminstration. C17-alpha alkylated anabolic/androgenic steroids can be hepatotoxic. Prolonged or high exposure may result in liver damage. In rare instances life threatening dysfunction may develop. It is advisable to visit a physician periodically during each cycle to monitor liver function and overall health. Intake of c17-alpha alkylated steroids is commonly limited to 6 - 8 weeks, in an effort to avoid escalating liver strain.
Side Effects (Cardiovascular):
Anabolic/androgenic steroids can have deleterious effects on serum cholesterol. This includes a tendancy to reduce HDL (good) cholesterol values and increase LDL (bad) cholesterol values, which may shift the HDL to LDL balance in a direction that favors greater risk of arteriosclerosis. The relative impact of an anabolic/androgenic steroid on serum lipids is dependent on the dose, route of administration (oral vs. injectable) type of steroid (aromatizable or non-aromatizable), and level of resistance to hepatic metabolism. Chlorodehydromethyltestosterone has a strong effect on the hepatic management of cholesterol due to its structural resistance to liver breakdown, non-aromatizable nature, and route of administration. Anabolic/androgenic steroids may also adversely affect blood pressure and triglycerides, reduce endothelial relaxation, and support left ventricular hypertrophy, all potentially increasing the risk of cardiovascular disease and myocardial infarction.
Side Effects (Testosterone Suppression):
All anabolic/androgenic steroids when taken in doses sufficient to promote muscle gain are expected to suppress endogenous testosterone production. Without the intervention of testosterone-stimulating substancestestosterone levels should return to normal within 1 - 4 months of drug secession. Note that prolonged hypogonadotrophic hypogonadism can develop secondary to steroid abuse, necessitating medical intervention.
Administration (Men):
A common clinical dose of chlorodehydromethyltestosterone is estimated to be 5mg per day; actual prescribing guidelines are unavailable. In the athletic arena, an effective oral daily dosage falls in the range of 15 - 40mg, taken in cycles lasting no more than 6 - 8 weeks to minimize hepatotoxicity. This level is sufficient for measurable increases in lean muscle mass and strength. This agent is most often applied as a pre-contest or cutting steroid for bodybuilding purposes, and is not viewed as an ideal bulking agent due to lack of estrogenicity. Athletes in sports where speed tends to be a primary focus also fine strong favor in chlorodehydromethyltestosterone, obtaining a strong anabolic benefit without having to carry around any extra water or fat weight.
Administration (Woman):
A common clinical dose of chlorodehydromethyltestosterone is estimated to be 1 - 2.5mg per day; actual prescribing guidelines are unavailable. In the athletic arena, women would commonly take a single 5mg tablet per day, taken in cycles lasting no more than 4 - 6 weeks to minimize hepatotoxicity. Virilizing effects are unlikely at this level of use. Much higher doses often used with female athletes in the former GDR doping program, but often to detriment of strong virilizing side effects.
Androgenic no data available
Anabolic >100
Standard Methyltestosterone (oral)
Chemical Names 4-chloro-17a-methyl-17b-hydroxyandrosta-1,4-dien-3-one
Estrogenic Activity none
Progestational Activity no data available (low)
Description:
Chlorodehydromethyltestosterone is a potent derivative of Dianabol. This oral steroid is structurally a cross between methandrostenolone and clostebol (4-chlorotestosterone), having the same base structure as Dianabol with the added 4-chloro alteration of clostebol. This alteration makes chlorodehydromethyltestosterone a milder cousin of Dianabol, the new steroid displaying no estrogenic and much less androgenic activity in comparison to its more famous counterpart. The anabolic activity chlorodehydromethyltestosterone is somewhat lower than that of Dianabol as well, but it does maintain a much more favorable balance of anabolic to androgenic effect. This means that at any given level of muscle-building activity, chlorodehydromethyltestosterone will be less likely to produce androgenic side effects.
Structural Characteristics:
Chlorodehydromethyltestosterone is a modified form of testosterone. It differs by: 1) the addition of a methyl group at carbon 17-alpha to protect the hormone during oral administration 2) the introduction of a double bond between carbons 1 and 2 (1-ene), which shifts the anabolic to androgenic ratio in favor of the former, and 3) the attachment of a chloro group at carbon 4, which inhibits steroid aromatization and reduces relative androgenicity.
Side Effects (Estrogenic):
Chlorodehydromethyltestosterone is not aromatized by the body, and is not measurably estrogenic. An anti-estrogen is not necessary when using this steroid, as gynecomastia should not be a concern even among sensitive individuals. Since estrogen is the usual culprit with water retention, this steroid instead produces a lean, quality look to the physique with no fear or excess subcutaneous fluid retention. This makes it a favorable steroid to use during cutting cycles, when water and fat retention are major concerns.
Side Effects (Androgenic):
Although chlorodehydromethyltestosterone is classified as an anabolic steroid, androgenic side effects are still possible with this substance. These may include bouts of oily skin, acne, and body/facial hair growth. Doses higher than normally prescribed are more likely to cause such side effects. Anabolic/androgenic steroids may also aggravate male pattern hair loss. Women are also warned of the potential virilizing effects of anabolic/androgenic steroids. These may include a deepening of the voice, menstrual irregularities, changes in skin texture, facial hair growth, and clitoral enlargement. Chlorodehydromethyltestosterone is not extensively metabolized by the 5-alpha reductase enzyme, so its relative androgenicity is not greatly altered by the concurrent use of finasteride.
Side Effects (Hepatotoxicity):
Chlorodehydromethyltestosterone is a c17-alpha alkylated compound. This alteration protects the drug from deactivation by the liver, allowing a very high percentage of the drug entry into the bloodstream following oral adminstration. C17-alpha alkylated anabolic/androgenic steroids can be hepatotoxic. Prolonged or high exposure may result in liver damage. In rare instances life threatening dysfunction may develop. It is advisable to visit a physician periodically during each cycle to monitor liver function and overall health. Intake of c17-alpha alkylated steroids is commonly limited to 6 - 8 weeks, in an effort to avoid escalating liver strain.
Side Effects (Cardiovascular):
Anabolic/androgenic steroids can have deleterious effects on serum cholesterol. This includes a tendancy to reduce HDL (good) cholesterol values and increase LDL (bad) cholesterol values, which may shift the HDL to LDL balance in a direction that favors greater risk of arteriosclerosis. The relative impact of an anabolic/androgenic steroid on serum lipids is dependent on the dose, route of administration (oral vs. injectable) type of steroid (aromatizable or non-aromatizable), and level of resistance to hepatic metabolism. Chlorodehydromethyltestosterone has a strong effect on the hepatic management of cholesterol due to its structural resistance to liver breakdown, non-aromatizable nature, and route of administration. Anabolic/androgenic steroids may also adversely affect blood pressure and triglycerides, reduce endothelial relaxation, and support left ventricular hypertrophy, all potentially increasing the risk of cardiovascular disease and myocardial infarction.
Side Effects (Testosterone Suppression):
All anabolic/androgenic steroids when taken in doses sufficient to promote muscle gain are expected to suppress endogenous testosterone production. Without the intervention of testosterone-stimulating substancestestosterone levels should return to normal within 1 - 4 months of drug secession. Note that prolonged hypogonadotrophic hypogonadism can develop secondary to steroid abuse, necessitating medical intervention.
Administration (Men):
A common clinical dose of chlorodehydromethyltestosterone is estimated to be 5mg per day; actual prescribing guidelines are unavailable. In the athletic arena, an effective oral daily dosage falls in the range of 15 - 40mg, taken in cycles lasting no more than 6 - 8 weeks to minimize hepatotoxicity. This level is sufficient for measurable increases in lean muscle mass and strength. This agent is most often applied as a pre-contest or cutting steroid for bodybuilding purposes, and is not viewed as an ideal bulking agent due to lack of estrogenicity. Athletes in sports where speed tends to be a primary focus also fine strong favor in chlorodehydromethyltestosterone, obtaining a strong anabolic benefit without having to carry around any extra water or fat weight.
Administration (Woman):
A common clinical dose of chlorodehydromethyltestosterone is estimated to be 1 - 2.5mg per day; actual prescribing guidelines are unavailable. In the athletic arena, women would commonly take a single 5mg tablet per day, taken in cycles lasting no more than 4 - 6 weeks to minimize hepatotoxicity. Virilizing effects are unlikely at this level of use. Much higher doses often used with female athletes in the former GDR doping program, but often to detriment of strong virilizing side effects.
You'll get a good run outta this at 60mg ED, I also throw in Proviron at 50mg a day as well.
Thank you for all you input. I cannot wait to order my gear and get rolling!
#9
Posted 09 October 2011 - 04:38 PM
As long as it's legit anavar, test E and 40mg anavar will be fine.
#10
Posted 02 February 2012 - 08:00 AM
Bump.. i have ran 40mgs var ed on a cycle a as much as 100mgs ed. Did not see any difference between 40 and 100.. especially if quality of var good..
#11
Posted 13 March 2012 - 04:04 AM
test prop, tren ace, mast , anavar 100mgs best shape of my life
#12
Posted 08 May 2012 - 09:13 AM
Yes, I realize the original post was from many months back, but I thought since it was revived I would comment anyway.......
Konan, it sounds good to me for what you are trying to accomplish. You should realize some decent gains (with good var) at around 40mgs var. Var is supposed to help burn visceral fat. Hmm, I need to run some myself....... You won't get the bloat you are worried about, but will get some decent strength from it. Creatine may help in addition.
Your test dose needs to be considered obviously, if you are concerned about blowing up too fast. Var is fairly popular with some of the lighter weight powerlifters who want to get stronger but not shoot up into the next weight class. Personally I think you'll like it if it's the real deal.
Better late than never..........
If you ran this var / test E cycle post up your thoughs on how it went.
Peace,
Mad_dawg
Konan, it sounds good to me for what you are trying to accomplish. You should realize some decent gains (with good var) at around 40mgs var. Var is supposed to help burn visceral fat. Hmm, I need to run some myself....... You won't get the bloat you are worried about, but will get some decent strength from it. Creatine may help in addition.
Your test dose needs to be considered obviously, if you are concerned about blowing up too fast. Var is fairly popular with some of the lighter weight powerlifters who want to get stronger but not shoot up into the next weight class. Personally I think you'll like it if it's the real deal.
Better late than never..........
If you ran this var / test E cycle post up your thoughs on how it went.
Peace,
Mad_dawg
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